Publication:
Heavy Metals and Kidney

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dc.contributor.authorOnan, Engin
dc.contributor.authorUlu, Sena Memnune
dc.contributor.authorGüngör, Özkan
dc.contributor.institutionOnan, Engin, Department of Nephrology, Başkent Üniversitesi, Ankara, Turkey
dc.contributor.institutionUlu, Sena Memnune, Faculty of Medicine, Bahçeşehir Üniversitesi, Istanbul, Turkey
dc.contributor.institutionGüngör, Özkan, Faculty of Medicine, Kahramanmaras Sütçü Imam Üniversitesi, Kahramanmaras, Turkey
dc.date.accessioned2025-10-05T14:45:30Z
dc.date.issued2024
dc.description.abstractMetals with a density of more than 5 g/cm3 are heavy metals, with more than 60 in nature. In acute and chronic exposure, they can damage many organs, such as the central nervous system, kidneys, skin, lungs, and heart. The most harmful ones to the kidney are lead, mercury, cadmium, and arsenic. They cause damage by creating some disorders in intracellular metabolic processes. The main types of kidney injury are acute tubular damage, proteinuria, and chronic kidney disease. This review discusses the basic properties of lead mercury cadmium and arsenic their nephrotoxicity mechanisms and studies about them. © 2024 Elsevier B.V., All rights reserved.
dc.identifier.doi10.5152/turkjnephrol.2024.22497
dc.identifier.endpage251
dc.identifier.issn26674440
dc.identifier.issue3
dc.identifier.scopus2-s2.0-85198706152
dc.identifier.startpage244
dc.identifier.urihttps://doi.org/10.5152/turkjnephrol.2024.22497
dc.identifier.urihttps://hdl.handle.net/20.500.14719/7095
dc.identifier.volume33
dc.language.isoen
dc.publisherTurkish Society of Nephrology
dc.relation.oastatusAll Open Access
dc.relation.oastatusGold Open Access
dc.relation.sourceTurkish Journal of Nephrology
dc.subject.authorkeywordsClinical Nephrology
dc.subject.authorkeywordsHeavy Metals
dc.subject.authorkeywordsKidney Disease
dc.titleHeavy Metals and Kidney
dc.typeReview
dcterms.referencesMetallerin Cevresel Etkileri I Tu Metalurji Ve Malzeme Muhendislii Bolumu, Jalili, Cyrus, Associations between exposure to heavy metals and the risk of chronic kidney disease: a systematic review and meta-analysis, Critical Reviews in Toxicology, 51, 2, pp. 165-182, (2021), Wang, Lin, Oxidative stress and apoptotic changes in primary cultures of rat proximal tubular cells exposed to lead, Archives of Toxicology, 83, 5, pp. 417-427, (2009), Satarug, Soisungwan, Cadmium and lead exposure, nephrotoxicity, and mortality, Toxics, 8, 4, pp. 1-42, (2020), Bravo, Yanauri, Mycophenolate mofetil administration reduces renal inflammation, oxidative stress, and arterial pressure in rats with lead-induced hypertension, American Journal of Physiology - Renal Fluid and Electrolyte Physiology, 293, 2, pp. F616-F623, (2007), Vaziri, Nosratola Dabir, Mechanisms of lead-induced hypertension and cardiovascular disease, American Journal of Physiology - Heart and Circulatory Physiology, 295, 2, pp. H454-H465, (2008), Ni, Zhenmin N., Lead exposure raises superoxide and hydrogen peroxide in human endothelial and vascular smooth muscle cells, Kidney International, 66, 6, pp. 2329-2336, (2004), Bennett, William Michael, Lead nephropathy, Kidney International, 28, 2, pp. 212-220, (1985), Ekong, E. B., Lead-related nephrotoxicity: A review of the epidemiologic evidence, Kidney International, 70, 12, pp. 2074-2084, (2006), Menke, Andy, Blood lead below 0.48 μmol/L (10 μg/dL) and mortality among US adults, Circulation, 114, 13, pp. 1388-1394, (2006)
dspace.entity.typePublication
local.indexed.atScopus
person.identifier.scopus-author-id55308728100
person.identifier.scopus-author-id55258968300
person.identifier.scopus-author-id8906634000

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